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In high-fat diet–induced NAFLD mouse models and human NAFLD liver samples, CAV1 deficiency correlated with enhanced oxidative stress, inflammation, and fibrosis, whereas supplementation with a CAV1 scaffolding domain peptide restored ferroptosis resistance and improved hepatic outcomes [409]

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These results are in keeping with those of a more recent case–case control study and meta-analysis that found no overall associations between asthma and plasma selenium in a large sample of subjects (45, 242)